Curriculum vitae

Forschungsschwerpunkte
  • Molekulare Zusammenhänge zwischen Stoffwechsel und Virulenz bei pathogenen Candida spp.
  • Die Rolle von Stoffwechselsignalen bei der Etablierung mikrobieller Gemeinschaften
  • Infektionsmodelle der Candidose
Wissenschaftlicher Werdegang
seit 2016 NWG-Leiterin, NWG Host Fungal Interfaces, ZIK Septomics, Friedrich-Schiller-Universität Jena
2014-2016 Assistant Professor-Research, University of Texas Health Science Center at Houston, Houston, TX, USA
2012-2016 Adjunct Faculty, University of Houston-Downtown, Houston, TX, USA
2013-2014 Senior Research Associate, University of Texas Health Science Center at Houston, Houston, TX, USA
2007-2013 Post-doctoral fellow, University of Texas Health Science Center at Houston, Houston, TX, USA
2007 Ph.D., State University of New York at Buffalo, Buffalo, NY, USA
2006-2007 Wissenschaftliche Mitarbeiterin (PostDoc), Ottawa Health Research institute, Centre for Cancer Research, Ottawa, ON, Canada
2002-2006 Wissenschaftliche Mitarbeiterin, State University of New York at Buffalo, Department of Oral Biology, Buffalo, NY, USA
2004 M.Sc., State University of New York at Buffalo, Buffalo, NY, USA
2004 Wissenschaftliche Mitarbeiterin, State University of New York at Buffalo, Physiology Department Buffalo, NY, USA
2001 M.Sc., Sofia University, Sofia, Bulgaria
Auszeichnungen · Ämter · wissenschaftliche Aktivitäten
seit 2021 Editor, Microbiology Spectrum
seit 2018 Editor, Frontiers in Microbiology
2014 Judge Travel Grant, Annual Biomedical Research Conference for Minority Students, San Antonio, TX, USA
2014 ISHAM Young Fellow Travel Award, American society for Microbiology, 12th ASM Conference on Candida and Candidiasis, New Orleans, LA, USA
2011 Office of the Dean of McGovern Medical School Postdoctoral Fellow Travel Grant, University of Texas Health Science Center Houston, Houston, TX, USA

Publikationen

Vylkova S, Sun JN, Edgerton M (2007) The role of released ATP in killing Candida albicans and other extracellular microbial pathogens by cationic peptides. Purinergic Signal 3(1-2), 91-97.
Vylkova S, Li XS, Berner JC, Edgerton M (2006) Distinct antifungal mechanisms: beta-defensins require Candida albicans Ssa1 protein, while Trk1p mediates activity of cysteine-free cationic peptides. Antimicrob Agents Chemother 50(1), 324-331.
Baev D, Rivetta A, Vylkova S, Sun JN, Zeng GF, Slayman CL, Edgerton M (2004) The TRK1 potassium transporter is the critical effector for killing of Candida albicans by the cationic protein, Histatin 5. J Biol Chem 279(53), 55060-55072.
Baev D, Rivetta A, Li XS, Vylkova S, Bashi E, Slayman CL, Edgerton M (2003) Killing of Candida albicans by human salivary histatin 5 is modulated, but not determined, by the potassium channel TOK1. Infect Immun 71(6), 3251-3260.
Dong J, Vylkova S, Li XS, Edgerton M (2003) Calcium blocks fungicidal activity of human salivary histatin 5 through disruption of binding with Candida albicans. J Dent Res 82(9), 748-752.

Links