(2024)
Inflammatory cytokine signalling in vulvovaginal candidiasis: a hot mess driving immunopathology.
Oxf Open Immunol 5(1),
iqae010.
(Review)
Dr. Axel Dietschmann
Adaptive Pathogenitäts-Strategien · Postdoktorand +49 3641 532-1366 axel.dietschmann@leibniz-hki.deThemenfelder
Publikationen
(2024)
Alpha1-antitrypsin impacts innate host-pathogen interactions with Candida albicans by stimulating fungal filamentation.
Virulence 15(1),
2333367.
(2024)
Nanobody-mediated neutralization of candidalysin prevents epithelial damage and inflammatory responses that drive vulvovaginal candidiasis pathogenesis.
mBio 15(3),
e0340923.
(2023)
Human TH17 cells engage gasdermin E pores to release IL-1α on NLRP3 inflammasome activation.
Nat Immunol 24(2),
295-308.
(2023)
Th2-dependent disappearance and phenotypic conversion of mouse alveolar macrophages.
Eur J Immunol 53(10),
e2350475.
(2023)
Th2-dependent STAT6-regulated genes in intestinal epithelial cells mediate larval trapping during secondary Heligmosomoides polygyrus bakeri infection.
PLoS Pathog 19(4),
e1011296.
(2022)
Phosphatidylinositol 3-kinase (PI3K) orchestrates Aspergillus fumigatus-induced eosinophil activation independently of canonical toll-like receptor (TLR)/C-type-lectin receptor (CLR) signaling.
mBio 13(4),
e0123922.
(2022)
Type I interferons during host-fungus interactions: Is antifungal immunity going viral?
PLOS Pathog 18(8),
e1010740.