(2016)
Dual-species transcriptional profiling during systemic candidiasis reveals organ-specific host-pathogen interactions.
Sci Rep 6,
36055.
Dr. Sascha Brunke
Mikrobielle Pathogenitätsmechanismen · Stellvertretender Abteilungsleiter +49 3641 532-1222 sascha.brunke@leibniz-hki.dePublikationen
(2016)
Candida albicans induces metabolic reprogramming in human NK cells and responds to perforin with a zinc depletion response.
Front Microbiol 7,
750.
(2016)
Widespread inter- and intra-Ddmain horizontal gene transfer of d-amino acid metabolism enzymes in eukaryotes.
Front Microbiol 7,
2001.
(2015)
Csr1/Zap1 maintains zinc homeostasis and influences virulence in Candida dubliniensis but is not coupled to morphogenesis.
Eukaryot Cell 14(7),
661-670.
(2015)
Of mice, flies - and men? Comparing fungal infection models for large-scale screening efforts.
Dis Model Mech 8(5),
473-486.
(2015)
Metal ions in host microbe interactions: The microbe perspective.
In: Nriagu JO, Skaar EP (eds.) Trace Metals and Infectious Diseases. 16, The MIT Press. Strüngmann Forum Reports.
ISBN: 9780262029193.
(2015)
Antifungal activity of clotrimazole against Candida albicans depends on carbon sources, growth phase, and morphology.
J Med Microbiol 64,
714-723.
(2014)
Adaptive prediction as a strategy in microbial infections.
PLOS Pathog 10(10),
e1004356.
(2014)
One small step for a yeast - Microevolution within macrophages renders Candida glabrata hypervirulent due to a single point mutation.
PLOS Pathog 10(10),
e1004478.
(2014)
Histidine degradation via an aminotransferase increases the nutritional flexibility of Candida glabrata.
Eukaryot Cell 13(6),
758-765.