(2017)
The Snf1-activating kinase Sak1 is a key regulator of metabolic adaptation and in vivo fitness of Candida albicans.
Mol Microbiol 104(6),
989-1007.
Dr. Sascha Brunke
Mikrobielle Pathogenitätsmechanismen · Stellvertretender Abteilungsleiter +49 3641 532-1222 sascha.brunke@leibniz-hki.dePublikationen
(2017)
Candida albicans Hap43 domains are required under iron starvation but not excess.
Front Microbiol 8,
2388.
(2016)
In vivo transcriptional profiling of human pathogenic fungi during infection: reflecting the real life?
PLOS Pathog 12(4),
e1005471.
(Review)
(2016)
Candida species rewired hyphae developmental programs for chlamydospore formation.
Front Microbiol 7,
1697.
(2016)
Virulence factors in fungal pathogens of man.
Curr Opin Microbiol 32,
89-95.
(Review)
(2016)
A novel hybrid iron regulation network combines features from pathogenic and non-pathogenic yeasts.
mBio 7(5),
e01782-16.
(2016)
Dual-species transcriptional profiling during systemic candidiasis reveals organ-specific host-pathogen interactions.
Sci Rep 6,
36055.
(2016)
Candida albicans induces metabolic reprogramming in human NK cells and responds to perforin with a Zinc depletion response.
Front Microbiol 7,
750.
(2016)
Widespread inter- and intra-Ddmain horizontal gene transfer of d-amino acid metabolism enzymes in eukaryotes.
Front Microbiol 7,
2001.
(2015)
Csr1/Zap1 maintains zinc homeostasis and influences virulence in Candida dubliniensis but is not coupled to morphogenesis.
Eukaryot Cell 14(7),
661-670.