(2020) The transcription factor Stp2 is important for Candida albicans biofilm establishment and sustainability. Front Microbiol 11, 794.
(2020) Catch the wave: Metabolomic analyses in human pathogenic fungi. PLOS Pathog 16(8), e1008757. (Review)
(2020) Clinical Candida albicans vaginal isolates and a laboratory strain show divergent behaviors during macrophage interactions. mSphere 5(4), e00393-20.
(2020) Metabolic modeling predicts specific gut bacteria as key determinants for Candida albicans colonization levels. ISME J [Epub ahead of print]
(2020) Ahr1 and Tup1 contribute to the transcriptional control of virulence-associated genes in Candida albicans. mBio 11(2), e00206-20.
(2020) Active neutrophil responses counteract Candida albicans burn wound infection of ex vivo human skin explants. Sci Rep 10(1), 21818.
(2020) Bloodstream infection due to Enterobacter ludwigii, correlating with massive aggregation on the surface of a central venous catheter. Infection 48(6), 955-958.
(2020) Bioflux analysis. Protocols ,
(2019) Role of amino acid metabolism in the virulence of human pathogenic fungi. Curr Clin Micro Rpt 6(9), 108-119. (Review)
(2017) Environmental pH modulation by pathogenic fungi as a strategy to conquer the host. PLOS Pathog 13(2), e1006149.
(2017) Phagosomal neutralization by the fungal pathogen Candida albicans induces macrophage pyroptosis. Infect Immun 85(2),
(2016) Robust Extracellular pH Modulation by Candida albicans during Growth in Carboxylic Acids. MBio 7(6),
(2014) Modulation of phagosomal pH by Candida albicans promotes hyphal morphogenesis and requires Stp2p, a regulator of amino acid transport. PLOS Pathog 10(3), e1003995.
(2011) The fungal pathogen Candida albicans autoinduces hyphal morphogenesis by raising extracellular pH. MBio 2(3), e00055.
(2009) Conservation and dispersion of sequence and function in fungal TRK potassium transporters: focus on Candida albicans. FEMS Yeast Res 9(2), 278-292.
(2008) Role of acetyl coenzyme A synthesis and breakdown in alternative carbon source utilization in Candida albicans. Eukaryot Cell 7(10), 1733-1741.
(2007) Histatin 5 initiates osmotic stress response in Candida albicans via activation of the Hog1 mitogen-activated protein kinase pathway. Eukaryot Cell 6(10), 1876-1888.
(2007) Human beta-defensins kill Candida albicans in an energy-dependent and salt-sensitive manner without causing membrane disruption. Antimicrob Agents Chemother 51(1), 154-161.
(2007) The role of released ATP in killing Candida albicans and other extracellular microbial pathogens by cationic peptides. Purinergic Signal 3(1-2), 91-97.
(2006) Distinct antifungal mechanisms: beta-defensins require Candida albicans Ssa1 protein, while Trk1p mediates activity of cysteine-free cationic peptides. Antimicrob Agents Chemother 50(1), 324-331.
(2004) The TRK1 potassium transporter is the critical effector for killing of Candida albicans by the cationic protein, Histatin 5. J Biol Chem 279(53), 55060-55072.
(2003) Killing of Candida albicans by human salivary histatin 5 is modulated, but not determined, by the potassium channel TOK1. Infect Immun 71(6), 3251-3260.
(2003) Calcium blocks fungicidal activity of human salivary histatin 5 through disruption of binding with Candida albicans. J Dent Res 82(9), 748-752.