Abstract
Candidemia, one of the most common causes of fungal bloodstream infection, leads to mortality rates up to 40% in affected patients. Understanding genetic mechanisms for differential susceptibility to candidemia may aid in designing host-directed therapies.
Leibniz-HKI-Autor*innen
Themenfelder
Identifier
doi: 10.1093/infdis/jiz206
PMID: 31241743