(2014)
Identification of Candida glabrata genes involved in pH modulation and modification of the phagosomal environment in macrophages.
PLOS One 9(5),
e96015.

Dr Sascha Brunke
Microbial Pathogenicity Mechanisms · Deputy head of department +49 3641 532-1222 sascha.brunke@leibniz-hki.dePublications
(2014)
Regulatory networks controlling nitrogen sensing and uptake in Candida albicans.
PLOS One 9(3),
e92734.
(2014)
Systematic phenotyping of a large-scale Candida glabrata deletion collection reveals novel antifungal tolerance genes.
PLOS Pathog 10(6),
e1004211.
(2014)
Immune evasion, stress resistance, and efficient nutrient acquisition are crucial for intracellular survival of Candida glabrata within macrophages.
Eukaryot Cell 13(1),
170-183.
(2014)
Microevolution of Candida albicans in macrophages restores filamentation in a nonfilamentous mutant.
PLOS Genet 10(12),
e1004824.
(2013)
Two unlike cousins: Candida albicans and C. glabrata infection strategies.
Cell Microbiol 15(5),
701-708.
(Review)
(2013)
Serial passaging of Candida albicans in systemic murine infection suggests that the wild type strain SC5314 is well adapted to the murine kidney.
PLOS One 8(5),
e64482.
(2013)
A core filamentation response network in Candida albicans is restricted to eight genes.
PLOS One 8(3),
e58613.
(2012)
Candida albicans scavenges host zinc via Pra1 during endothelial invasion.
PLOS Pathog 8(6),
e1002777.
(2012)
Candida albicans dimorphism as a therapeutic target.
Expert Rev Anti Infect Ther 10(1),
85-93.
(Review)