(2018) FHR5 binds to laminins, uses separate C3b and surface-binding sites, and activates complement on malondialdehyde-acetaldehyde surfaces. J Immunol 200(7), 2280-2290.
Prof. Dr. Peter F. ZipfelInfection Biology · Head International Leibniz Research School · Speaker +49 3641 532-1301 email@example.com
Main Research Areas
- Immune escape of human pathogenic micro-organisms
- Infection-associated function of the complement system
- Genetic susceptibility for infections
|Since 2000||Professor for infection biology, Friedrich-Schiller-University Jena (FSU Jena)|
|Since 2000||Head of the department infection biology, Leibniz-Institute for Natural Product Research and Infection Biology – Hans-Knöll-Institute Jena|
|1999||Nontenured professor, University Hamburg|
|1993||Habilitation in immunology and molecular biology, University Hamburg|
|1989-2000||Group head at the Bernhard-Nocht-Institute for tropical medicine Hamburg|
|1989||Visiting associate, Laboratory of Immunoregulation, National Institutes of Health, Bethesda, Maryland, USA|
|1985-1988||Postdoc, Laboratory of Immunoregulation, National Institutes of Health, Bethesda, Maryland, USA, funded by the German Academic Exchange Service (DAAD)|
|1984||PhD as Dr. rer. nat., University Bremen|
|1980-1985||Research assistant, University Bremen|
|1980||Diploma in biology, University Bremen|
Awards · Appointments · Scientific Activities
|Since 2009||Member at the editorial board of molecular immunology, frontiers in innate immunity|
|2009||Excellence award from the Deutsche Gesellschaft für Hygiene und Mikrobiologie|
|Since 2008||Representative of the graduate school International Leibniz Research School for Microbial and Biomolecular Interactions (ILRS)|
|2008||EFIS lecture award of the European Federation of Immunological Societies|
|Since 2007||Principal investigator of the excellence graduate school Jena School for Microbial Communication (JSMC)|
|2007||Heinz Spitzbart award of the European Society for Infectious Diseases in Obstetrics and Gynaecology (ESIDOG)|
|Since 2006||Principal investigator of the graduate school International Leibniz Research School for Microbial and Biomolecular Interactions (ILRS)|
|2007-2011||President of the European Complement Network|
|2005 – 2012||Board member of the European Complement Network|
|2004||Thuringian research award|
|Since 2002||Deputy Director of the Leibniz-Institute for Natural Product Research and Infection Biology – Hans-Knöll-Institute Jena|
|Since 2000||Project leader for infection biology, L2|
|1999-2001||Member at the editorial board of experimental and clinical immunology, thrombosis and haemostasis, section editor molecular immunology
Member of the European Working Party on the Genetics of Complement mediated Kidney Diseases, Deutsche Gesellschaft für Hygiene und Mikrobiologie (DGHM), Deutsche Gesellschaft für Immunologie, Gesellschaft für Genetik, Gesellschaft für Nephrologie
(2018) Membranoproliferative glomerulonephritis and C3 glomerulopathy in children: Change in treatment modality? A report on a case series. Clin Kidney J 11(4), 479-490.
(2018) Reply to kang and brooks: Comment on the interpretation of binding of Pra1, the fungal immune evasion protein from Candida albicans to the human C3 and on the conformational changes of C3 upon activation: Kang and brooks optimization of biolayer-interfero. Mol Immunol 101, 638-639.
(2017) Direct Binding of the pH-Regulated Protein 1 (Pra1) from Candida albicans Inhibits Cytokine Secretion by Mouse CD4(+) T Cells. Front Microbiol 8, 844.
(2017) Anaphylatoxins Activate Ca2+, Akt/PI3-Kinase, and FOXO1/FoxP3 in the Retinal Pigment Epithelium. Front Immunol 8, 703.
(2017) Atypical hemolytic uremic syndrome and C3 glomerulopathy: conclusions from a "Kidney Disease: Improving Global Outcomes" (KDIGO) Kidney Int 91(3), 539-551.
(2017) Successful discontinuation of eculizumab under immunosuppressive therapy in DEAP-HUS. Pediatr Nephrol 32(6), 1081-1087.
(2017) Factor H Binds to Extracellular DNA Traps Released from Human Blood Monocytes in Response to Candida albicans. Front Immunol 7, 671.
(2017) Binding of Candida albicans to Human CEACAM1 and CEACAM6 Modulates the Inflammatory Response of Intestinal Epithelial Cells. mBio 8(2), pii: e02142-16.
(2017) Moss-Produced, Glycosylation-Optimized Human Factor H for Therapeutic Application in Complement Disorders. J Am Soc Nephrol 28(5), 1462-1474.